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| Product | Description | |
|---|---|---|
| ZL006 | ?98% (HPLC). Group: Fluorescence/luminescence spectroscopy. |  |
| ZL006 | ZL006 is a potent inhibitor of nNOS/PSD-95 interaction, and inhibits NMDA receptor -mediated NO synthesis. Uses: Scientific research. Group: Signaling pathways. CAS No. 1181226-02-7. Pack Sizes: 10 mM * 1 mL; 5 mg; 10 mg; 25 mg; 50 mg; 100 mg. Product ID: HY-100456. |  |
| ZL006 | ZL006 is a potent inhibitor of nNOS/PSD-95 interaction. In mice subjected to mCao and reperfusion, pretreatment with Zl006 attenuated the ischemia-induced increase in nnos-PsD-95 complex levels, and in neurons treated with glutamate this drug ameliorated nmDar-dependent nitric oxide synthesis. Synonyms: ZL006; ZL-006; ZL 006; Benzoic acid, 4-[[(3,?5-dichloro-2-hydroxyphenyl)?methyl]?amino]?-2-hydroxy-. CAS No. 1181226-02-7. Molecular formula: C14H11Cl2NO4. Mole weight: 328.15. |  |
| nNOS - PSD-95 Interaction Inhibitor, ZL006 (5-(3,5-dichloro-2-hydroxybenzylamino)-2-hydroxybenzoic Acid) | A cell-permeable and blood-brain barrier-permeant aminosalicylic acid compound that selectively disrupts nNOS and PSD-95 interaction uponnMDAR activation both in primary murine cortical neurons (0.8- and 2.8-fold increase from basal level with or without 30min 10uM ZL006 pretreatment) in vitro and in mice (0.4- and 1.9-fold increase from basal level with or without inhibitor pretreatment 15min before ischemia; 1.5mg/kg i.v.) in vivo without affecting nNOS activity or non-nNOS-mediated nMDAR EPSCs (excitatory postsynaptic currents). Effectively reduce ischemia-caused brain tissue damage in both mice and rats when applied 1h after blood reperfusion following ishemia (60% and 50% reduction of infarction, respectively; 1.5mg/kg i.v.). Group: Biochemicals. Grades: Highly Purified. Pack Sizes: 10mg. US Biological Life Sciences. |  Worldwide |
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